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In years gone by, women would rely on the calendar on the wall to work out when their next menstrual cycle might occur. They would look to physical signs to tell them when they might be ovulating, and therefore when they’d be most likely to fall pregnant.
A miscarriage is a devastating event. Those who experience them are suddenly and unexpectedly robbed of the promise of new life and the dream of an expanded family. The emotional toll can be even greater if conception was delayed, or if fertility treatments were required to achieve a pregnancy.
It is increasingly clear that genetics alone do not explain risks of developing allergies, and that environmental exposures before and around birth can program individuals to increased or decreased risk of allergies. Restricted growth before birth in preclinical studies appears to protect the offspring against allergic responses. However, whether prenatal growth predicts subsequent risk of allergy in humans is unclear. Many studies in humans use birth weight as a measure of fetal growth, but do not correct for gestational age, so effects of premature birth may confound those of fetal growth.
Professor David Haig from Harvard University presented the 2019 Lloyd Cox Memorial Lecture on the topic: When fetal and maternal interests collide – an evolutionary conundrum.
Michelle Clarke from the Robinson Research Institute traveled to Ljubljana, Slovenia in May to attend the ESPID conference, including plenary/invited talk sessions, e-poster viewing sessions and themed oral presentation sessions.
Children with type 1 diabetes find it difficult to adhere to their drug routines during school holidays and weekends. Holiday distractions cause a 20% reduction in adherence to taking medications that assist managing their condition and other associated conditions, which may have serious consequences for their health.
Soon Wei Wong from the Robinson Research Institute travelled to Davos, Switzerland to attend the 13th World Immune Regulation Meeting where he presented a poster on “A Novel Role for ZEB2 in the Human CD4+ T cell Compartment”.